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Levels of amyloid-beta protein fluctuate over the day and night/sleep-wake cycle in the brain of a mouse genetically engineered to express the human amyloid precursor protein (top). The brain of a sleep-restricted mouse (bottom left) shows more amyloid plaques compared with the brain from a mouse that did not undergo sleep restriction (bottom right). Dark deposits represent amyloid plaques, which are associated with Alzheimer's disease in humans. Credit: David Holtzman and Jae-Eun Kang

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